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. 2014 Jun 27;13:159. doi: 10.1186/1476-4598-13-159

Table 3.

Gene expression analysis of DCK in a set of primary MCL samples obtained from patients before and after araC-based therapies

Sample at diagnosis Source ∆CT (DCK-GAPDH) Therapy Sample at relapse Disease-free survival (months) Source ∆CT (DCK-GAPDH) Difference in ∆CT between R and D samples
D1
PBMC
3.4
A*
R1
12
PBMC
3.7
+0.3
D2
PE***
3.3
A
R2
10
PE***
5.3
+2.0
D3
FFPE
0.1
A
R3
5
FFPE
1.3
+1.2
D4
FFPE
1.7
B
R4
4
FFPE
3.5
+1.8
D5
PBMC
1.4
A
R5
7
PBMC
2.2
+0.8
D6
PBMC
4.1
B**
R6
3
PBMC
3.9
−0.2
D7
FFPE
1.3
B
R7
13
FFPE
3.5
+2.2
D8
FFPE
2.0
A
R8
25
FFPE
1.8
−0.2
D9
PBMC
1.9
B
R9
N/A
PBMC
3.3
+1.4
D10 PBMC 2.3 A R10 N/A PBMC 1.5 −0.8

*A = alternation of R-CHOP and R-araC (2 g/m2, 2 doses a 24 h).

**B = Nordic protocol (alternation of R-MaxiCHOP and R-araC (2-3 g/m2, 4 doses a 12 h).

***PE pleural effusion (CD19-sorted).

Samples from relapsed patients were obtained at diagnosis (D1-D8) and at lymphoma relapse after failure of araC-based therapies (R1-R8). Samples from refractory patients were obtained from primary araC-resistant MCL patients before (D9-D10) and 14 days after (R9-R10) administration of high-dose araC. Real-time RT-PCR was used to determine changes in DCK expression.