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. Author manuscript; available in PMC: 2014 Jul 15.
Published in final edited form as: Hypertension. 2011 Aug 1;58(3):431–438. doi: 10.1161/HYPERTENSIONAHA.111.172338

Figure 3.

Figure 3

Effect of Pin1 inhibition on endothelial NO synthase (eNOS) Ser116 phosphorylation. Experiments were performed in rat aortic endothelial cells in the absence or presence of juglone without (A) or with (B) the eNOS agonist vascular endothelial growth factor (VEGF). Representative Western blots showing eNOS Ser116 phosphorylation and densitometry for ratio of eNOS Ser116 phosphorylation: eNOS as a percentage of control. Actin was used as a loading control. Results are expressed as mean±SEM (n=5 independent experiments). *P<0.05 vs “0,” +P<0.05 vs previous time point.