Figure 1.

Potential mechanisms by which prior exercise reduces postprandial lipemia. Aerobic exercise has been shown to increase FA substrate utilization during metabolic studies presumably in skeletal muscle and liver. Increased hepatic FA oxidation and decreased de novo lipogenesis are thought to decrease VLDL-TG synthesis and secretion. Increased clearance of postprandial triglycerides through upregulation of LPL expression and activity has also been proposed in skeletal muscle and to a lesser extent in adipose tissue.