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. 2014 May 7;14:322. doi: 10.1186/1471-2407-14-322

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Copyright © 2014 Xue et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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RNPC1a regulated p53, p21, E-cadherin and vimentin in breast cancer cell. (A) RNPC1a positively regulated p21, while there was no significantly correlation was found between RNPC1a and wtp53 in MCF-7 cells. (B) RNPC1a positively regulated p21, while negatively regulated mutp53 in MDA-MB-231 cells. (C) RNPC1a positively regulated E-cadherin, while negatively regulated Vimentin in MDA-MB-231 cells. (D) RNPC1a positively regulated of E-cadherin, while there was no significantly correlation was found between RNPC1a and Vimentin in MCF-7 cells. The fold change of RNPC1a is shown below each lane. Arbitrarily set at 1.0 in control cells. The intensity of the bands was determined using densitometric analysis.