Table 1.

Patient characteristics.

No	TLE Durati on (years)	Pathologically proven MTS	Pathology	Side	Risk Factor	ECoG	Reason for ECoG	Age at Surgery (years)	Follow- up Duration (years)
1	35	Y	AHS	L	closed head injury	Y	temporal vs. extratemporal	53.6	2.8
2	59	Y	AHS	L	febrile seizure (18 months), minor closed head injury	N		61	4.5
3	5	Y	AHS	L	febrile seizure, heroin abuse with multiple overdoses	Y	R vs L	45.8	2.5
4	39	Y	AHS	L	febrile seizure	N		43.9	2.6
5	59	Y	AHS	L	febrile seizure	N		60.7	4.3
6	31	Y	AHS	L	closed head injury; fever	N		51.6	4.4
7	18	Y	AHS	L	meningitis (18 months)	N		38.4	2.9
8	10	Y	AHS	R	febrile seizure	Y	R vs. L	15.0	4.6
9	47	Y	AHS	R	Fetal ethyl alcohol syndrome	Y	temporal vs. extratemporal	47.5	5.3
10	28	Y	CA, AHS	R	None	N		49.0	4.0
11	28	N	nonspecific CA (Corpora amylacea) no pathology on hippocamp	L	febrile seizure (24 months)	N		30.4	3.2
12	37	N	No specimen received	L	febrile seizure	N		39.3	2.6
13	27	N	FCD	L	None	N		28.7	2.6
14	12	N	Non-diagnostic specimen (too small)	R	closed head injury	N		27.3	3.7
15	47	N	gliosis, FCD	R	febrile seizure, cerebral palsy	N		48.3	4.0
16	6	N	nonspecific changes	R	childhood sz related to fluid management after intussusceptions	N		28.6	4.2
17	46	N	ill-defined hippocampal structure with suggestion of mild neuronal loss	R	childhood meningitis	Y	R vs. L temporal vs. extratemporal	53.1	4.3
18	8	N	No hippocampus received	R	closed head injury	Y	R vs. L	43.7	3.5
19	8	N	—	R	closed head injury	N		27.7	5.4
20	29	N	temporal pole granulomas	R	physical abuse	Y	temporal vs. extratemporal	45.1	4.1

Table notation: ECoG, electrocorticography; TLE, temporal lobe epilepsy; L, left; R, right; CA, corpora amylacea; AHS, Ammon’s horn sclerosis; FCD, focal cortical dysplasia; Y, Yes; N, No.