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. 2014 May 15;7(8):963–976. doi: 10.1242/dmm.014449

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© 2014. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 2. — Loss of dohh-1 function in C. elegans leads to early embryonic lethality. (A) Representation of the C. elegans dohh-1 locus and the gk398 deletion allele. (B) C. elegans DOHH-1 protein contains 6 EZ-type HEAT repeat domains. (C–G) Differential interference contrast microscopy images (DIC) of dohh-1 homozygous mutant animals show normal larval development (F–G). Adult animals exhibit wild-type germ lines (C), but defective embryos (arrows in D,E,H). (I–J) DIC of heterozygous dohh-1(gk398)/mIn1[mIs14 dpy-10(e128)] II animals.