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. 2014 Aug;350(2):301–312. doi: 10.1124/jpet.114.215855

Fig. 5.

Fig. 5.

PKC and protein tyrosine kinase do not contribute significantly to nerve injury–induced increase in NMDAR activity of spinal dorsal horn neurons. (A and B) Original recordings (A) and mean changes (B) in the amplitude of evoked NMDAR-EPSCs and AMPAR-EPSCs of lamina II neurons in SNL rat spinal cord slices treated with vehicle (n = 16 neurons) or 10 μM chelerythrine (n = 10 neurons) 3 weeks after nerve injury. (C) Summary data show the amplitude of evoked NMDAR-EPSCs and AMPAR-EPSCs of lamina II neurons in SNL rat spinal cord slices treated with vehicle (n = 11 neurons), 100 μM genistein (n = 12 neurons), or 1 μM PP2 (n = 8 neurons). Error bars represent the S.E.M.