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. Author manuscript; available in PMC: 2015 Apr 1.

Published in final edited form as: Life Sci Space Res (Amst). 2014 Feb 17;1:80–88. doi: 10.1016/j.lssr.2014.02.006

Effects of NF-κB in the regulation of genomic instability induced by cytoplasmic irradiation in ρ⁺ SAE cells. A. Representative image of NF-κB p65 immunocytochemistry staining of ρ⁺ and ρ⁰ SAE cells. B. Quantification of NF-κB p65 by mean gray value of ρ⁺ and ρ⁰ SAE cells. Levels of NF-κB p65 were significantly increased post cytoplasmic irradiation with ten α particles in ρ⁺ SAE cells but not in ρ⁰ SAE cells. C. Representative image of iNOS immunocytochemistry staining of ρ⁺ and ρ⁰ SAE cells. D. iNOS expression were significantly increased induced by cytoplasmic irradiation in ρ⁺ SAE cells, however, ρ⁰ SAE cells showed no response.

#, P<0.01, *, P<0.05 versus the control group. Bars indicate ± SD. Results were repeated in three other experiments. In each experiment, 100 cells were scored.

Effects of NF-κB in the regulation of genomic instability induced by cytoplasmic irradiation in ρ⁺ SAE cells. A. Representative image of NF-κB p65 immunocytochemistry staining of ρ⁺ and ρ⁰ SAE cells. B. Quantification of NF-κB p65 by mean gray value of ρ⁺ and ρ⁰ SAE cells. Levels of NF-κB p65 were significantly increased post cytoplasmic irradiation with ten α particles in ρ⁺ SAE cells but not in ρ⁰ SAE cells. C. Representative image of iNOS immunocytochemistry staining of ρ⁺ and ρ⁰ SAE cells. D. iNOS expression were significantly increased induced by cytoplasmic irradiation in ρ⁺ SAE cells, however, ρ⁰ SAE cells showed no response.

#, P<0.01, *, P<0.05 versus the control group. Bars indicate ± SD. Results were repeated in three other experiments. In each experiment, 100 cells were scored.