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. 2014 Mar 8;210(4):611–618. doi: 10.1093/infdis/jiu140

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© The Author 2014. Published by Oxford University Press on behalf of the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs licence (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial reproduction and distribution of the work, in any medium, provided the original work is not altered or transformed in any way, and that the work is properly cited. For commercial re-use, please contact journals.permissions@oup.com.

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Figure 1. — Chromosome 2 map of the 7 SNPs genotyped in the full family-based study. Chromosome 2 location at 191,894,306–192,015,925 bp (2q32.2-q32.3) of STAT4 (121.62 kb, and the translated product comprises 748 amino acids) is presented. The 24 exons are shown in black, introns in gray, and the promoter region and 3′ UTR in light gray. Locations of the STAT4 start and stop codons are indicated by arrows. Out of the 7 SNPs, the SNP that is not significantly associated with pulmonary tuberculosis in the full familial sample is in italics, and the 3 SNPs significantly associated with pulmonary tuberculosis in the combined familial and case-control samples are shown in bold. Below, pairwise R² values for all pairs of SNPs are given as percentages, and shading from white to black indicates intensity, from an R² of 0 to 1. Abbreviation: SNP, single-nucleotide polymorphism.