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. 2013 Oct 25;63(8):1313–1324. doi: 10.1136/gutjnl-2012-304058

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This is an Open Access article distributed in accordance with the terms of the Creative Commons Attribution (CC BY 3.0) license, which permits others to distribute, remix, adapt and build upon this work, for commercial use, provided the original work is properly cited. See: http://creativecommons.org/licenses/by/3.0/

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Features of fatty acid ethyl ester-induced acute pancreatitis induced by palmitoleic acid (POA)/ethanol. Mice received two intraperitoneal injections of ethanol (1.35 g/kg) in combination with POA at 0, 10, 20, 80 and 150 mg/kg, and mice were sacrificed 24 h after first injection. (A) Representative H&E images of histology slides from pancreas of mice treated with ethanol with POA. (B) Ethanol with POA (150 mg/kg) caused alveolar membrane thickening and inflammatory infiltration of the lung but did not induce any significant damage to liver, kidney or heart. Effects of ethanol with POA on (C) serum amylase, (D) Pancreatic trypsin activity and (E) Pancreatic myeloperoxidase activity (normalised). *p<0.05 compared to POA alone group. Values are mean±SE of 4–6 mice. Magnification: ×200.