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. Author manuscript; available in PMC: 2015 Sep 1.
Published in final edited form as: Biochim Biophys Acta. 2014 May 9;1840(9):2878–2885. doi: 10.1016/j.bbagen.2014.05.004

Fig. 6.

Fig. 6

Inactivation of RAS/cAMP-PKA pathway is required for glucose starvation-induced Hxt1 turnover. (A) Yeast cells (WT) coexpressing Hxt1-GFP in combination with either empty vector or Ras2Val19 were grown as described in Fig. 2A. Membrane fractions were immunoblotted with anti-GFP antibody. (B) Yeast cells (WT and bcy1Δ) expressing Hxt1-GFP were grown as described in Fig. 2A Membrane fractions were immunoblotted with anti-GFP antibody. Actin was served as loading control.