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. 2014 Aug;196(15):2789–2797. doi: 10.1128/JB.01783-14

FIG 5.

FIG 5

Pretreatment with proteinase K nearly abolishes the ability of the ΔpgfS strain to bind to collagen and invade HCAEC. OMZ175 and its ΔpgfS and CpgfS derivatives were treated with 3 μg ml−1 of proteinase K for 30 min, and their abilities to bind to collagen (A) and invade HCAEC (B) were assessed. Results are expressed as the ratio of binding by proteinase K-treated cells to that by untreated cells. *, P < 0.05.