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. 2014 Jul 28;12(8):4291–4310. doi: 10.3390/md12084291

Figure 3.

Figure 3

(A) Representative photomicrographs showed p-Akt (A1–D1), p-Bad (A2–D2) and caspase-3 (A3–D3) immunohistochemical staining of the temporal lobe in all experimental groups. As shown, low p-Akt, p-Bad and caspase-3 staining in the cerebral cortex was observed in the sham group rats, while strong p-Akt, p-Bad and caspase-3 staining could be seen in the SAH group and SAH + vehicle groups. After ATX administration, the immunoreactivity of p-Akt and p-Bad in the cerebral cortex were further increased following SAH, whereas the immunoreactivity of caspase-3 was significantly downregulated; (B) Quantitative analysis of the p-Akt (b1), p-Bad (b2) and caspase-3 (b3) immunohistochemical staining in the temporal lobe in all experimental groups. Values are expressed as the mean ± SEM. *** p < 0.001, ** p < 0.01, * p < 0.05, ns p > 0.05.