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. 2014 Jun 11;5(13):5153–5164. doi: 10.18632/oncotarget.2095

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Copyright: © 2014 Liu et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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A. The genes down-regulated in response to MTA1 overexpression are highly enriched on gut development and differentiation; B. MTA1 promotes the proliferation of cancer cells; C. The genes up-regulated in response to MTA1 knockdown show the highest enrichment score on the functional annotation cluster for development and differentiation; D. Transient knock down of MTA1 using siRNA causes the floating and death of HCT116 colon cancer cells; E. Western blot analysis showing the effect of MTA1 siRNA (si-MTA1) on HCT116 cells; F. At 72 h after transfection, the number of survival si-MTA1 transfected cells was 2/3 that of control cells (P<0.05).