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. Author manuscript; available in PMC: 2014 Aug 29.
Published in final edited form as: Biol Psychiatry. 2013 Aug 31;75(3):238–247. doi: 10.1016/j.biopsych.2013.07.020

Table 7. Decomposition of the Correlations (rPh) between RTV and EEG Measures into Genetic (rph-a) and Environmental (rph-e) Components and the Overlap in Genetic (rG) and Environmental (rE) Factors between Them.

RTV

rPh rph-a rph-e rG rE
Source PhV .49 (.27 to.69)a .34 (.1 to.58)a .15 (0 to .38) .66 (.14 to .99)a .32 (−.10 to.63)
Source AmM −.34 (−.55 to −.13)a −.18 (−.44 to .14) −.17 (−.27 to −.01)a −.31 (−.93 to .37) −.38 (−.66 to −.01)a
Channel PhV .18 (.17 to .44)a .20 (.18 to .33)a −.01 (−.37 to .34) .54 (.22 to .99)a −.02 (−.11 to .71)
Channel PhM .34 (.13 to .44)a .18 (−.27 to .47) .15 (−.03 to .48) .40 (−.1 to .99) .29 (−.06 to .58)

rG, rE: The correlation between genetic and nonshared environmental factors. These are combined with the standardized estimates of the traits (Table 4) to establish the genetic (rph-a) and unique environmental (rph-e) contributions to the total phenotypic correlation (rPh).

EEG, electroencephalographic; AmM, mean amplitude; AmV, variability in amplitude; PhM, mean phase; PhV, phase variability; RTV, reaction time variability.

a

Significant estimates.