Table 4.
Group | Fat diet | Plasma lipid peroxidation (nmole of MDA/mL) | Erythrocyte membrane lipid peroxidation (nmole of MDA/mL) | Liver tissue lipid peroxidation (nmole of MDA/mL) |
---|---|---|---|---|
Control (non-diabetic) | SO | 10.71 ± 0.414# | 6.64 ± 0.44# | 1.62 ± 0.34# |
Diabetic groups | SO | 27.21 ± 0.35 | 15.23 ± 0.26 | 3.4 ± 0.62 |
SO+ 0.5 % ESA NEa | 5.34 ± 0.25* | 3.26 ± 0.17* | 1.42 ± 0.81* | |
SO+ 0.25 % ESA NEb | 4.8 ± 0.33* | 2.83 ± 0.91* | 1.28 ± 0.33* | |
SO+ 0.5 % ESA CEc | 5.2 ± 0.32* | 4.23 ± 0.42* | 1.73 ± 0.63* | |
SO+ 0.25 % ESA CEd | 6.12 ± 0.27* | 4.4 ± 0.31* | 2.5 ± 0.24* | |
[MDA] comparison among diet groups | ALX(SO)>CON(SO)>0.25 % ESA CE>0.5 % ESA NE>0.5 % ESA CE >0.25 % ESA NE |
Values are mean ± SEM, n = 6
(ALX-D) treated diabetic group fed with sunflower oil
SO sunflower oil; BGO bitter gourd oil; ESA α-eleostearic acid; NE nanoemulsion
*Marked results show mean values for the BGO NE treated experimental groups that are statistically different (p < 0.05) than the alloxan (ALX-D) treated diabetic group fed with sunflower oil diet.
#Marked results show mean values for the non diabetic control group that are statistically different (p < 0.05) than the alloxan
a0.5 % ESA NE is present in 1 % BGO NE; b0.25 % ESA NE is present in 0.5 % BGO NE; c0.5 % ESA CE is present in 1 % BGO CE; d0.25 % ESA CE is present in 0.5 % ESA CE