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. 2004 Jun;186(11):3531–3538. doi: 10.1128/JB.186.11.3531-3538.2004

FIG. 1.

FIG. 1.

Comparison of the PlcR (A) and PapR (B) sequences of various B. anthracis, B. cereus, and B. thuringiensis strains. Ba, B. anthracis (a superscript asterisk, no superscript, and a superscript number sign indicate the Sterne, Ames, and RA3R strains, respectively); Bc, B. cereus ATCC 14579; LM112.3, B. cereus LM112.3; Bt1, Bt13, and Bt32, B. thuringiensis Bt1, Bt13, and Bt32, respectively; 26, 17, 5, and 45, B. thuringiensis strains 26, 17, 5, and 45, respectively; 1, B. thuringiensis 407 Cry− strain. Bt1, Bt13, and Bt32 were isolated from the environment for this study and have not been serotyped yet. Sequences corresponding to the sequences of the hemolytic strains are enclosed in boxes. The sequences were aligned by using Megalign (DNASTAR). In panel A, the nonsense mutations in PlcR are indicated by stars. The other mutations thought to be responsible for the loss of hemolytic and lecithinase activities are circled. The nucleotide sequences of plcR of B. thuringiensis 407 Cry−, B. cereus ATCC 14579, B. anthracis Sterne, and B. anthracis Ames have been described previously (1, 30, 42).