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. Author manuscript; available in PMC: 2014 Sep 8.
Published in final edited form as: J Neurochem. 2003 Feb;84(4):655–668. doi: 10.1046/j.1471-4159.2003.01571.x

Fig. 6.

Fig. 6

Inhibition of caspase activity with z-VAD-fmk does not prevent loss of cell viability in PC12 cultures exposed to either palmitic or stearic fatty acid complexed with BSA (2 : 1). (a) Cell viability was assessed after 12 and 24 h of exposure to either stearic and palmitic acid in the presence and absence of 100 μm z-VAD-fmk as indicated. Data represent the mean ± SD from three independent experiments performed in triplicate. (b) Caspase-3-like activity in cellular extracts of PC12 cells exposed to stearic and palmitic in the presence and absence of z-VAD-fmk (100 μm). Time points for stearic (15 h) and palmitic (12 h) acid were chosen to correspond to the peak of induced caspase-3-like activity. Data represent the mean ± SD for two independent experiments.