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. 2014 Aug 23;2:98. doi: 10.1186/s40478-014-0098-6

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© Prins et al.; licensee BioMed Central Ltd. 2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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CCL2 mRNA in astrocytes and CCL2 induced microglial proliferation . Primary WM rat astrocytes produce significantly more CCL2 mRNA upon treatment with bz-ATP than GM astrocytes (A). Primary rat microglia significantly increased their proliferation rate upon treatment with CCL2. This effect was abolished when the cells were co-incubated with a CCR2 antagonist (B). Immunofluorescent double labelling study showed colocalization between BM28 and CCR2 in post mortem human hippocampal GML (C). * p < 0,05.