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. Author manuscript; available in PMC: 2014 Sep 11.
Published in final edited form as: Genes Brain Behav. 2011 Aug 15;10(7):805–816. doi: 10.1111/j.1601-183X.2011.00720.x

Figure 1. Conditional knockdown of the NR2B subunit of the NMDA receptor in NR2B deficient (KO) mice.

Figure 1

A significant reduction in protein levels of NR2B was observed in (a) dorsomedial prefrontal cortex (dmPFC), (b) dorsal hippocampus (HPC), (c) dorsolateral striatum (DLS), and (d) nucleus accumbens (NAc) in NR2B deficient (KO) mice in comparison with wildtype (WT), CAM, and CRE mice (n = 4-7 males/genotype). NR1 expression levels in NR2B deficient mice were also significantly reduced in each of these regions. No significant changes in NR2A subunit expression levels were observed in any of the genotypes. * = differs from WT, CRE, CAM