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. 2014 Sep 12;9(9):e107068. doi: 10.1371/journal.pone.0107068

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© 2014 Dhasmana et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(A) Internalization of BaP in to cell through AHR, metabolic conversion to BPDE and interaction of BPDE with DNA. (B) Preferential binding of TiO₂NP with AHR. TiO₂ NP bound to AHR blocks the internalization of BaP, preventing its metabolic conversion to BPDE and finally avoiding DNA damage.