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. Author manuscript; available in PMC: 2015 Oct 1.
Published in final edited form as: Clin Neurophysiol. 2014 Mar 12;125(10):2070–2078. doi: 10.1016/j.clinph.2014.01.035

Table 1.

Subject demographic and clinical information from 17 chronic stroke survivors.

Subject ID Sex Age, yr Months Post Stroke Antigravity Spasticity Index Lesion Location Paretic Side/Hand Dominance
2 F 52 33 0.125 L BG R / R
3 F 52 302 0.750 R MCA L / R
4 M 56 68 0.375 L BG - lateral thalamic R / R
5 F 62 226 0.375 R BG L / R
6 M 52 45 0.500 R Carotid dissection L / L
7 M 51 39 0.188 R MCA L / L
8 M 66 99 0.625 R Periatrial WM - L Medial Temporal L / R
9 M 48 16 0.313 L IC - BG - centrum semi-ovale R / L
10 M 62 225 0.375 L MCA R / R
11 F 54 84 0.750 R BG L / R
12 F 61 154 0.375 R IC L / R
13 M 56 35 0.563 R IC - corona radiata L / R
14 F 58 301 0.438 L Carotid artery R / R
15 F 64 24 0.625 R BG L / R
16 M 52 35 0.625 L Thalamic R / L
17 F 71 265 0.375 L BG - centrum semiovale R / R
18 F 44 16 0.438 R IC - thalamic - corona radiata L / R

Abbreviations: MCA, middle cerebral artery; BG, basal ganglia; WM, white matter; IC, internal capsule.

Notes: Subject 1 was excluded because of comorbid conditions. Notch filtering was used on both sides of subject 13 due to 60 Hz noise.

It should be noted that while subject 8 had a chronic focal lesion in the right periatrial white matter and a chronic microhemmorhage in the left medial temporal lobe, this subject presented with lateralized spasticity in both the upper and lower limbs on the left side. The left sided spasticity was presumably an outcome of the right-sided white matter lesion.