FIGURE 7. Proposed model for the local induction of APC-like neutrophils under the influence of microbe-responsive unconventional T-cells.
(A) Upon pathogen clearance, neutrophils release microbial metabolites into the microenvironment where they stimulate local or freshly recruited unconventional T-cells to release pro-inflammatory cytokines. (B) In the presence of unconventional T-cell derived mediators like GM-CSF, IFN-γ and TNF-α, bystander neutrophils acquire the capacity to act as APCs for tissue resident and/or newly arriving effector and memory CD4+ and CD8+ T-cells. Activated neutrophils may also gain access to inflamed draining lymph nodes and prime T-cell responses in secondary lymphoid tissues (not depicted).