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. Author manuscript; available in PMC: 2015 Oct 1.
Published in final edited form as: J Immunol. 2014 Aug 29;193(7):3549–3558. doi: 10.4049/jimmunol.1401138

Figure 4. CD4, CD8, NK, and γδ cells are the major source of CXCL1-mediated IL-17A and IL-17F production and rCXCL1 rescues T cell subsets in cxcl1−/− mice in response to PMS.

Figure 4

Intracellular IL-17A (A) and IL-17F (B) in gated CD4+, CD8a, γδ, and NK1.1 cells from lung and spleen of WT, cxcl1−/− mice, and cxcl1−/− mice after rCXCL1/KC administration at 24 h post-PMS was analyzed by flow cytometry. The results are expressed as mean ± SE from three independent experiments (n=5-8/group; *, p<0.05; **, p<0.01; ***, p<0.001).