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. 2014 Jul 10;5(14):5819–5831. doi: 10.18632/oncotarget.2171

Fig. 6. Celastrol and its derivatives induced degradation and inhibited phosphorylation of HSP90/CDC37 client proteins in orthotopic HCC patient-derived xenografts.

Fig. 6

Representative Western Blot results are shown for client proteins and their phosphorylation levels in two random tumors (T1 and T2) of HCC-3 xenografts after 3 weeks of treatment with each compound and saline control. GAPDH was used as a loading control.