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. 2014 Oct 1;31(19):1625–1631. doi: 10.1089/neu.2014.3372

FIG. 2.

FIG. 2.

Representative cranial imaging of children at initial radiographic evaluation and after development of HH is shown. The first child suffered bilateral subdural hematomas (1a and 1b; white arrow) and showed development of bilateral HH (1c; black-in-white arrow). The second child also had bilateral injury (2a and 2b; white arrow) and showed predominately unilateral parenchymal insult, as shown on the apparent diffusion coefficient map (2c; black-in-white arrow). The third child had predominate right-sided subdural blood (3a; white arrow) and, despite emergent decompressive craniectomy, suffered HH as depicted on postoperative computed tomography and magnetic resonance imaging (diffusion-weighting imaging; DWI), as shown in 3b and 3c, respectively (black-in-white arrow). The fourth child showed bifrontal subdural blood with other bleeding patterns, particularly noted by traumatic subarachnoid hemorrhage and contusions (4a; white arrow) and rapidly developed HH (4b; black-in-white arrow), as corroborated on DWI (4c; black-in-white arrow). SDH, subdural hematoma; HH, hemispheric hypotensity.