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. Author manuscript; available in PMC: 2015 Jun 1.
Published in final edited form as: Appetite. 2014 Mar 6;77:113–121. doi: 10.1016/j.appet.2014.02.019

Fig. 1.

Fig. 1

Diagram of the study hypotheses. The first hypothesis tests the relationship between PROP status and intake of sweets and savory-fats at a palatable buffet meal. It is predicted that nontaster children will have increased intake of savory-fats in part due to decreased oral fat discrimination (not measured). It is also predicted that taster children will have increased intake of sweets at the meal, in part mediated by increased sweet taste perception (not measured). The second hypothesis tests the relationship between TAS2R38 genotype and intake of sweets and savory-fats. We predict that children with the AVI (nontaster) genotype will consume more savory-fats at the meal, while children with the PAV (taster) genotype will consume more sweets at the meal.