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. 2014 Sep 23;9(9):e107776. doi: 10.1371/journal.pone.0107776

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© 2014 Xie et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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(A) DMH1 treatment showed no effect on the protein expressions of PP2A-a, PP2A-b, and PP2A-c subunits. (B) DMH1 treatment inhibited PP2A activity in L6 cells. **P<0.01 vs control. (C) PP2A activator forskolin (40 µM) inhibited DMH1-induced Akt activation in L6 cells. **P<0.01 vs control. ## P<0.01 vs DMH1.(D–E) Forskolin (40 µM) showed no effects on p-AMPK level in L6 cells.