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. 2014 Sep 24;8:317. doi: 10.3389/fnbeh.2014.00317

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Copyright © 2014 Prast, Schardl, Schwarzer, Dechant, Saria and Zernig.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Cocaine CPP-induced EGR1 expression is reversed by social interaction preferentially in D1-MSNs. Shown are means ± s.e.m.'s of EGR1-immunoreactive nuclei expressed as percent of dynorphin-labeled cells, presumably D1-MSNs (open bars) vs. D2-MSNs labeled with the dopamine D2 receptor (grid-pattern bars) for the accumbens corridor regions, the AcbCl, and the CPu. Panel (A) (red bars) shows the percent of activated D1-MSNs and D2-MSNs for the COCAINE group, whereas panel (B) (green) those for the SOCIAL group. ^*p < 0.05; ^**p < 0.01; ^***p < 0.001.