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FSTL1 decreased neuron apoptosis after MCAO was dependent on its receptor, DIP2A. DIP2A-siRNA increased neuron apoptosis (A, B) and increased neurological deficits (C) in FSTL1 treated rats after MCAO. n=6 for each group. & p<0.05 vs. MCAO+Vehicle, @ p<0.05 vs. MCAO+FSTL1+Scramble-siRNA. Scale bars=100 μm.
