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. 2014 Sep 18;12:38. doi: 10.1186/1476-7120-12-38

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© Mada et al.; licensee BioMed Central Ltd. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Colour coded strain rate curved M-modes (top) and strain curves (middle) in 2 myocardial segments both showing post-systolic shortening (PSS). Left panel: physiologic PSS in an apical septum of a normal heart. Peak systolic strain exceeds −18% and PSS is less than 20% of systolic shortening. Right panel: pathologic PSS in a partially scarred segment post infarction. Peak systolic strain is reduced to −10.1% while post systolic strain is increased to 39% of systolic strain. AVC - Aortic valve closure; MVO - mitral valve opening. (modified from Voigt et al. [22], with permission).