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. 2014 Aug 22;1(8):605–617. doi: 10.1002/acn3.91

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© 2014 The Authors. Annals of Clinical and Translational Neurology published by Wiley Periodicals, Inc on behalf of American Neurological Association.

This is an open access article under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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IFNB-1b and 25(OH)D-regulated genes are additionally associated with regulating the count of Gd-enhancing lesions. (A) Venn diagram depicting the overlap between genes that are associated with Gd-enhancing lesions, 25(OH)D or IFNB-1b. (B) Genes whose induction is associated with a reduction of Gd lesion count and which are upregulated through 25(OH)D and IFNB-1b. (C) Genes whose induction is associated with a reduction of Gd lesion count and which are downregulated through 25(OH)D and IFNB-1b. (D) Genes whose induction is associated with an increase of Gd lesion count and which are upregulated through 25(OH)D and IFNB-1b. (E) Genes whose induction is associated with an increase in Gd lesion count and which are downregulated through 25(OH)D and IFNB-1b. IFNB-1b, interferon beta-1b; 25(OH)D, 25-hydroxyvitamin D.