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Proposed TLR signaling mechanism by CS14K: a hypothetical model of TLR4 signaling, pathway activated by fusion protein, in a MyD88-independent fashion. Inhibition of p65 movement to the nucleus by CS14K prevents the action of NF-κB while activation of IRF-3 and STAT-1 leads to elevated levels of NO, IL-6, and TNF-a, which could inhibit the growth of parasites.
