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B. pertussis-induced enhanced Slc26a4 levels are STAT6 independent. Lung loads at 4 dpi with B. pertussis WT were similar in BALB/c and Stat6−/− mice (n = 4) (A). qPCR revealed no significant difference in infection-induced Slc26a4 upregulation (B). Expression levels were normalized to control PBS-treated BALB/c mice and displayed as mean ± SEM. *, P < 0.05 compared with control PBS-treated mice.
