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. 2014 Sep 27;14:723. doi: 10.1186/1471-2407-14-723

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© Ji et al.; licensee BioMed Central Ltd. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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ERK pathway in the Kiss-1 regulated motility of colorectal cancer cells. A. Left: The effect of Kiss-1 and Kiss-1R knockdown on the migration ability of HT115 cells treated withKisspeptin-10 compared with control group (no treatment). Right: Overall changes in resistance at the eighth hour * p < 0.05 vs the respective control group at the specific time point. B. Left: Cells in two different experimental settings (control and Kisspeptin-234) during 8 hours after wounding. Right: Overall changes in resistance at the eighth hour. C. Left: The mean trace of the cells in two groups of experiments (control and ERK inhibitor) 8 hours after wounding. Right: Overall changes in resistance at the eighth hour. D. Left: Cell responses after wounding (control and ERK inhibitor + Kisspeptin-10) during 8 hours after wounding. Right: Overall changes in resistance at the eighth hour. E. Left: Cells responses in different experimental settings, namely control and ERK inhibitor + Kisspeptin-234, over an 8 hours period after wounding. Right: Overall changes in resistance at the eighth hour.