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. 2014 Aug;141(15):2984–2992. doi: 10.1242/dev.109827

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© 2014. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 7. — Expression of Lgl-GFP in the germline rescues the slmb polarity phenotype. (A) A slmb^9H4-17 germline clone egg chamber showing the mislocalisation of Staufen (Stau, red) in a cloud in the centre of the oocyte. (B) A typical slmb^9H4-17 germline clone egg chamber expressing Lgl-GFP. Lgl-GFP (green) localises normally to the posterior cortex of the oocyte and rescues the posterior localisation of Stau. (C) The frequency of Stau localisation phenotypes in slmb^9H4-17 germline clones with and without expression of Lgl-GFP. (D) Model presenting Slmb as the functional counterpart of C. elegans PAR-2. Both proteins are required for the posterior exclusion of the aPKC complex and for the recruitment of Par-1, and both can be rescued by overexpressing Lgl. Border cells are shown in yellow.