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Shock absorption via Src-mediated focal adhesion/nonmuscle cytoskeleton regulation is defective in old aortas: model of how changes in stiffness generated by cross-bridge attachment and remodeling of focal adhesions and nonmuscle cortical cytoskeleton can be regulated by a PP2-sensitive mechanism present in young but not old aortas. FA, focal adhesion; NMC, nonmuscle cytoskeleton; MLCK, myosin light chain kinase.
