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. 2004 Jun;15(6):2782–2793. doi: 10.1091/mbc.E04-03-0179

Figure 6.

Figure 6.

Suppression of ovulation defects by a mutation of unc-60B ADF/cofilin. (A) Micrographs of wild-type (a-d) or unc-60 (r398) (e-h) on agar plates after control (a and e), CeTMI,II (RNAi) (b and f) CeTMI,II,III,IV (RNAi) (c and g), or pat-10 (RNAi) (d and h) treatment. Bar, 0.5 mm. (B and C) The myoepithelial sheath (B) or the spermatheca (C) of dissected gonads from wild-type (a, c, e, and g) or unc-60 (r398) (b, d, f, and h) worms after control treatment (a and b), CeTMI,II (RNAi) (c and d), CeTMI,II,III,IV (RNAi) (e and f), or pat-10 (RNAi) (g and h) were stained by tetramethylrhodamine-phalloidin to visualize F-actin in the myoepithelial sheath. Note that intensely stained aggregates in c, e, f, and g were present in the oocytes not in the sheath cells due to aberrant cell division. Bars, 50 μm (B) or 10 μm (C).