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. 2014 Jul 31;5(17):7610–7624. doi: 10.18632/oncotarget.2282

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Copyright: © 2014 Li et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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TW02 and TW06 cells transiently expressing (a) vector control, or miR-148a (1μg); (b) vector control, or miR-148a (1μg); and control (siCtrl) or miR-148a inhibitor (anti-miR, 50 pmole), were used to perform transwell cell migration assay. Migrated cells of 10 microscopic fields from 3 independent, duplicate experiments were counted. (c) Wound-healing assays were performed using similar conditions as in (b). The gap area (wound) was measured and analyzed by ImageJ at the indicated time.

TW02 and TW06 cells transiently expressing (a) vector control, or miR-148a (1μg); (b) vector control, or miR-148a (1μg); and control (siCtrl) or miR-148a inhibitor (anti-miR, 50 pmole), were used to perform transwell cell migration assay. Migrated cells of 10 microscopic fields from 3 independent, duplicate experiments were counted. (c) Wound-healing assays were performed using similar conditions as in (b). The gap area (wound) was measured and analyzed by ImageJ at the indicated time.