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. Author manuscript; available in PMC: 2015 Nov 1.
Published in final edited form as: Int J Eat Disord. 2014 Jun 25;47(7):773–783. doi: 10.1002/eat.22321

Table 2.

Indices of Fit for Nested ACE Models Examining the Etiology of Thin-Ideal Internalization by Age and Pubertal Development.

Model −2lnL df Δχ2 (df) p AIC BIC
Age Models
Full Moderation 2854.44 1024 -- -- 806.44 −1786.42
Linear Moderation 2855.83 1027 1.39 (3) 0.71 801.83 −1795.14
No Moderation 2857.06 1030 2.62 (6) 0.85 797.06 1803.94
Puberty Models
Linear Moderation 2822.34 1011 -- -- 800.34 −1754.98
No Moderation 2829.67 1014 7.33 (3) 0.06 801.67 1760.71

Note. −2lnL = minus 2 times the log likelihood, Δχ2 = change in chi-square (−2lnL) from the full moderation model; AIC = Akaike information criteria, BIC = Bayesian information criterion; Mod = Moderator; DE = Disordered Eating; A=additive genetic effects; C = Shared environmental effects, E = Nonshared environmental effects. Best-fitting models, as determined from non-significant chi-square and lowest AIC and BIC values, are indicated with bold text. In the “Full” moderation model, genetic, shared environmental, and nonshared environmental estimates are allowed to vary both linearly and quadraticaly across levels of the moderator (i.e., age or pubertal development). In the “Linear” moderation model, genetic, shared environmental, and nonshared environmental estimates are allowed to vary linearly across levels of the moderator. In the “No Moderation” model, genetic, shared environmental, and nonshared environmental estimates are constrained to be equal across all age or pubertal groups. A full moderation model was not calculated for pubertal groups since quadratic moderation is not possible when examining only two groups.