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. 2014 Nov 1;28(21):2348–2360. doi: 10.1101/gad.250902.114

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© 2014 Alcid and Tsukiyama; Published by Cold Spring Harbor Laboratory Press

This article is distributed exclusively by Cold Spring Harbor Laboratory Press for the first six months after the full-issue publication date (see http://genesdev.cshlp.org/site/misc/terms.xhtml). After six months, it is available under a Creative Commons License (Attribution-NonCommercial 4.0 International), as described at http://creativecommons.org/licenses/by-nc/4.0/.

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Figure 3. — Chromatin remodeling factors repress ASlncRNAs. (A–D) Representative strand-specific RNA-seq data in Δswr1 (A), Δitc1 (B), tet-STH1 (C), and tet-INO80 (D) mutants. Blue and purple signals denote Watson and Crick strand transcripts, respectively. The direction of the coding gene transcription is shown at the bottom of each panel. (E–H) ASlncRNA meta analyses. The ratio of ASlncRNA levels in in Δswr1 (E), Δitc1 (F), tet-STH1 (G), and tet-INO80 (H) mutants relative to wild-type cell levels is shown in log₂ scale. The black lines represent the average signals, and the colored lines represent the RNA signals of the top 25% and bottom 25% changes in the mutants.