Table 2. Effects of Site-Specific Intra-Preoptic Administration of the Alpha₁ Receptor Agonist, Methoxamine on Hindbrain Glucoprivic Inhibition of Rostral Preoptic GnRH Nerve Cell Fos Expression in Steroid-Primed Ovariectomized Female Rats.

Treatment Group	Cell Counts^a
Treatment Group	GnRH-ir Neurons	GnRH-/Fos-ir Neurons
A. Rostral Preoptic Area (Pre-Treatment of aCSF vs. ME)
aCSF + SAL_CV4	81 ± 9	53 ± 6
aCSF + 5TG _CV4	68 ± 7	29 ± 3^b
ME + 5TG_CV4	75 ± 9	26 ± 3^b
B. Anteroventral Periventricular Nucleus (Pre-Treatment of aCSF vs. ME)
aCSF + SAL_CV4	93 ± 10	64 ± 7
aCSF + 5TG_CV4	85 ± 9	25 ± 3^b
ME + 5TG_CV4	78 ± 9	50 ± 6
C. Median Preoptic Nucleus (Pre-Treatment of aCSF vs. ME)
aCSF + SAL_CV4	69 ± 8	40 ± 5
aCSF + 5TG_CV4	80 ± 9	24 ± 3^b
ME + 5TG_CV4	73 ± 9	21 ± 3^b

Mean number ± S.E.M. for n=5 rats/group

p<0.05 compared to aCSF + SAL group

Table 2. Effects of Site-Specific Intra-Preoptic Administration of the Alpha1 Receptor Agonist, Methoxamine on Hindbrain Glucoprivic Inhibition of Rostral Preoptic GnRH Nerve Cell Fos Expression in Steroid-Primed Ovariectomized Female Rats.