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. Author manuscript; available in PMC: 2014 Nov 6.
Published in final edited form as: Anal Bioanal Chem. 2011 Aug 13;401(7):2165–2175. doi: 10.1007/s00216-011-5311-x

Fig. 4.

Fig. 4

Quantitative electrophoretic profiling of MQ-mediated glutathionylation in HT-29 cells and Pr–SSG of brain tissues and blood in AD–Tg/WT mice by using CGE-LIF. a Electropherograms of Pr–SSG from HT-29 cells labeled with Dylight 488 maleimide. Trace 1 demonstrates Pr–SSG from MQ-treated HT-29 cells. Traces 2 and 3 indicate MQ untreated HT-29 cell and MQ-treated HT-29 cells without deglutathionylation, respectively. b Electropherograms of Pr–SSG in cerebrum of AD–Tg/WT mice at 5 months. c Electropherograms of Pr–SSG from three anatomical positions (cerebrum, cerebellum, and hippocampus) in brain tissue and blood sample of AD–Tg mouse. d Electropherograms of Pr–SSG from three anatomic brain regions and blood of AD-WT mouse. 1, 2: Dylight 488 maleimide. All the experimental conditions are the same as in Fig. 2. Top trace is offset in the Y axis for the clarity