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. Author manuscript; available in PMC: 2015 Nov 1.
Published in final edited form as: J Neurochem. 2014 Aug 6;131(4):521–529. doi: 10.1111/jnc.12824

Figure 2.

Figure 2

Treatment with EPO inhibited superoxide anion production. Cerebral microvessels of hph1 mice demonstrated increased production of superoxide anions (* P<0.05 vs. WT, n=8), and the increased superoxide anion production was sensitive to L-NAME (30 µM, # P<0.05 vs. hph1, n=4).Treatment with EPO significantly attenuated superoxide anion production in cerebral microvessels of hph1 mice. Incubation with L-NAME (30 µM) did not affect superoxide anion production in cerebral microvessels of hph1 mice treated with EPO.