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. 2014 Nov 10;207(3):375–391. doi: 10.1083/jcb.201404016

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© 2014 Ogun and Zallocchi

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).

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Figure 9. — Clarin-1 MOs show dysregulation in the number of ribeye b boutons in neuromasts. (A–L) Representative experiments of 3 dpf neuromasts from Tg(bnr3c:mGFP) immunostained for ribeye b and MAGUK in control MOs (A–C), clarin-1 (Clrn1) MOs (D–F), MOs + cRNA (G–I), or cRNAΔCT (J–L). (insets) High magnification of juxtaposed ribeye b/MAGUK boutons. Bars: (main images) 5 µm; (insets) 2 µm. (M) Quantification of ribeye b punctate per neuromast. Results from each independent experiment are represented by open (control animals) or filled (treated animals) circles. Mean for each treatment is depicted as a line with error bars representing SEM. N, number of independent experiments (neuromasts analyzed), n > 14. **, P < 0.01; ***, P < 0.001.