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. 2014 Jun 10;5(3):76. doi: 10.1186/scrt465

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Copyright © 2014 Tan et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Effect of exosomes in G₁phase of the cell cycle after APAP- or H₂O₂-induced injury in hepatocytes. Expression of NF-κB (p65 and p50) and phosphorylated STAT3 was determined by immunoblotting after 24-hour treatment in APAP- or H₂O₂-injury models. The relative densities of NF-κB and phosphorylated STAT3 bands were normalized to actin in the same samples.