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Impact of reverse mutations Kir2.1 L222I and Kir2.3 I214L on cholesterol dependence of Kir–PIP2 interactions.
A: The time courses of Kir2.1 WT and Kir2.1-L222I rundown in response 10 μM neomycin in control and cholesterol depleted cells. B: The time courses of Kir2.3 WT and Kir2.3-I214L rundown in response 1 μM neomycin in control and cholesterol depleted cells (all data show means + SEM, n = 5–10 cells per condition).
