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Gαs overexpression in ASM enhances relaxation of airways from TG1 mice. A: contraction response to acetylcholine (ACh) was unaltered in TG1 airways. B: extent of relaxation of acetylcholine-contracted rings to the β-agonist isoproterenol (Iso) was greater in TG1 airways overexpressing Gαs than in NTG littermates. C: relaxation response to the bitter taste receptor agonist quinine (Qui) was not altered in TG1 mice. Values are means ± SE; n = 4 mice in each group. *P < 0.01 vs. NTG.
