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. 2014 Nov 19;10:272. doi: 10.1186/s12917-014-0272-x

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© Brommer et al.; licensee BioMed Central Ltd 2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Histopathology of the interface of the PSBs and the ISL. Transverse sections of a diseased and a non-diseased Friesian horse, haematoxylin and eosin staining. Note the multifocal to coalescing inflammation of the ISL which is characterized by abundant fibroblasts, lymphocytes and plasma cells in the diseased horse. The transition from ligament to bone (arrow) is very irregular in the diseased horse and compared to the non-diseased horse, there is a decrease of surface area where ligament tissue merges to bone in the diseased horse. Remaining adjacent bone shows increased osteoclastic bone resorption (osteoclasts marked with asterisks). Bone marrow was hypercellular due to invasion of lymphocytes and plasma cells.