Skip to main content
. Author manuscript; available in PMC: 2015 Mar 1.
Published in final edited form as: Kidney Int. 2013 Dec 4;85(3):611–623. doi: 10.1038/ki.2013.462

Figure 8. Effects of AMP-activated protein kinase (AMPK) activation in lipid accumulation in kidney and liver.

Figure 8

Quantitative analysis of triglycerides, long-chain nonesterified fatty acid (NEFA), short-chain NEFA, cholesteryl esters, and phosphatidylcholine levels in liver (a, c, e, g, i, respectively) and in the kidney (b, d, f, h, j, respectively) in mice fed a standard diet (STD), a high-fat diet (HFD), or HFD + AICAR. Values are means ± s.e.m. N = 6 in each group. Statistical analyses were performed by one-way analysis of variance (ANOVA) followed by Newman-Keuls, *P ≤ 0.05 versus mice on STD and #P ≤ 0.05 versus mice on HFD. AICAR, 5-aminoimidazole-4-carboxamide-1-β-d-ribofuranoside.